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GSH acts as a copper shuttle Beyond its role in redox cycling, GSH is a key regulator of intracellular copper trafficking

The key pathophysiological downstream effect of GBA mutations is decreased glucocerebrosidase (GCase) activity and lysosomal dysfunction and experimental studies have shown a reciprocal relationship of GCase activity and -synuclein aggregation [191, 192]

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178,179 Conversely, low potassium levels can contribute to arterial calcification, high blood pressure, and stroke